Researchers have uncovered a surprising new role for the HSL protein: beyond breaking down fat, it also works inside the nucleus of fat cells to keep them functioning properly. When HSL is missing, fat tissue doesn’t expand as expected— instead, it shrinks, leading to lipodystrophy. This unexpected discovery helps explain why both obesity and fat-loss disorders share similar health risks, and it opens up fresh paths for understanding metabolic diseases at a time when obesity affects billions worldwide.
  • Valmond@lemmy.worldEnglish
    3·
    4 days ago

    Interesting but not directly linked to classic obesity whatever the article tries to suggest in the end, or so I understand it.

    • chosensilence@pawb.socialEnglish
      2·
      3 days ago

      i am thankful my glp-1 provides me significant relief from and control of metabolic disorder. having lost 150 lbs so far it wouldn’t have been possible had i felt the hunger and compulsion to eat. my brain does not see foods the same way as i did prior to my shots. i truly do consider myself lucky and know this will likely be lifelong but am fine with that. i have something that can be treated medically and it is a chronic condition.

      if this could at least help shed the weight it could be useful for those on a glp-1 during a stall…? not sure.

      • Valmond@lemmy.worldEnglish
        2·
        3 days ago

        I’m all for scientific research and advancement (good on you by the way 👍🏻) but less so on click baity headlines.

        If I understood it’s directed at genetic disorders linked to the proteins controlling fat storage and usage, and some interesting findings has been, well, found.

        Cheers and hang in there!